EPR-Proton Qubits’ Role in Evolution and Age-Related
Journal Title: Physical Science & Biophysics Journal - Year 2017, Vol 1, Issue 1
Abstract
Earth’s surface acquired necessary life-giving volatile elements carbon, nitrogen, and sulfur from a collision with a Mercury-like planetary embryo ~ 4.4 billion y ago. Icy comets containing hydrocarbons collided with a cooling prebiotic Earth to create impact reactive environments that via classical anthropic causality introduced primordial “ribozyme-like” RNA complexes which could duplicate a few molecular units/24 hrs. Random classical processes introduced energetically accessible duplex RNA segments containing keto─amino (─NH2) hydrogen bonds, where hydrogen bonded amino protons encountered quantum uncertainty limits, Δx Δpx ≥ ħ/2. This introduced probabilities of EPR-arrangements, ketoamino―(entanglement) → enol−imine, where reduced energy product protons are each shared between two different indistinguishable sets of electron lone-pairs belonging to enol oxygen and imine nitrogen on opposite genome strands. Product protons participate in entangled quantum oscillations at ~4×1013 s −1 (~ 4800m s−1) between near symmetric energy wells in decoherence-free subspaces until measured, δt << 10−13 s, in a genome groove, ~12 or 22Å, by selected Grover’s quantum bio-processors. Analyses imply entanglement origins of the triplet code, 43 codons, specifying ~ 22 L-amino acids. Entanglement resources provided a sequence of ~ 12 incremental entanglement-enabled improvements to genome fitness, of the form: RNA-ribozyme → genetic code origin → RNA-protein → DNA-protein. An EPR-entanglement algorithm explains “probabilistic” genomic growth over the past ~ 3.6109 y from duplex RNA–ribozyme segments, into a DNA double helix of ~ 6.8109 bp. Entangled proton “qubit pairs” are the smallest “measurable” genetic informational unit, specifying evolution instructions with “measured” quantum information. This EPR-entanglement model accurately predicts microsatellite evolutionary distributions in rat and human genomes. Consistent with preserving “wild-type” gene pool viability, Huntington’s and other age-related human diseases are phenotypically expressed by Grover’s quantum processors, measuring quantum informational content of entangled proton qubits occupying a “threshold limit”.
Authors and Affiliations
W. Grant Cooper*
Keywords
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