ZIKA VIRUS and MICROCEPHALY

Journal Title: ESTÜDAM Halk Sağlığı Dergisi - Year 2016, Vol 1, Issue 1

Abstract

Zika virus was first identified in a Rhesus monkey in Zika forest of Uganda in 1947. The first human cases were detected in Uganda and United Republic of Tanzania in 1952. The virus caused outbreaks in French Polinesia and some of Pasific Islands in 2013, immediately after outbreaks occured in Brasil and Columbia dating from 2015. Transmisson of the virüs primarily happens through the bite of an infected Aedes species mosquito (Aedes aegypti, Ae. Albopictus), the other less common ways are transmission during pregnancy, through sex, blood transfusion, contamination with body fluids (sweat, tear, saliva, urine) and direct contact. Congenital Zika Syndrome may contain manifestations like microcephaly, craniofacial disproportion, spasticity, seizures, irritability, peduncle abnormalities, arthrogryposis multiplex congenita, abnormal ocular findings, abnormal radiological screening findings like cerebral calcifications, cortical disfuntion, ventriculomegaly. Microcaphaly is the most remarkable clinical outcome of these and scientists continue their studies about this relationship.

Authors and Affiliations

Emrah Atay, Selma Metintaş

Keywords

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  • EP ID EP287707
  • DOI -
  • Views 118
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How To Cite

Emrah Atay, Selma Metintaş (2016). ZIKA VIRUS and MICROCEPHALY. ESTÜDAM Halk Sağlığı Dergisi, 1(1), 46-57. https://europub.co.uk/articles/-A-287707