Effect of Diet on Evolutionary Obstetrics
Journal Title: Interventions in Gynaecology and Women’s Healthcare - Year 2018, Vol 3, Issue 1
Abstract
Diet and health are both related directly to the reproductive functions of a female in that nutrient intake effects exactly what hormones are produced and at what amounts. According literature women of Western societies produce more progesterone and estradiol from their ovaries leading to high rates of breast cancer in the West [1]. Another facet to the Western diet is foods that are rich in sugar thus contributing to the blood sugar level and ultimately affecting resistance to insulin. This resistance to insulin, which is called diabetes, may lead to many problems of the reproductive function of women. Obesity, insulin resistance, and hyperinsulinemia are all common of women in the West and lead to oligomenorrhea, amenorrhea, and chronic anovulation [2]. Regions pay big role into nutrition in the sense that different areas have abundance of different types of foods, a bulk of North America simply gets absorbed into the “western” diet, meaning increased intake of unhealthy food due to increased availability of fast food. Since these female populations are adopting nutritious lifestyles leading to obesity, insulin resistance, and hyperinsulinemia, these newly developing populations have women who are unable to reproduce as well, thus making this “developing society” not “develop.” The current research is supported with case studies demonstrating insulin levels, SHBG, and testosterone levels of women who are of south Asian descent living in affluent societies such as the United Kingdom [3]. The evidence provided portrayed that though there were two type of women living in the same society, women from south Asia that were adopting the new Western lifestyles were more susceptible to having high levels of insulin and insulin resistance, thus proving that the societies that are westernizing is leading to reproductive failure of the women. What this portrays is that high levels of obesity, insulin resistance, and insulin itself leads populations in transition to have reduced fecundity in women and based on the examples provided, it is a logical conclusion [4].
Authors and Affiliations
Satyajit Patra, Shakti Sagar
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