Pathways Independent of Contamination may Produce Burn Wound Infections
Journal Title: Biomedical Journal of Scientific & Technical Research (BJSTR) - Year 2018, Vol 8, Issue 5
Abstract
Background: A plethora of evidence suggest pathways independent of contamination may produce microbial growth and infections. Objective: To prove that pathways independent of contamination may produce burn wound infections. Methods: We searched the keywords burn wound infections in Google scholar and pub med for articles and their references published in English from 2000 to 2017. We then applied the probability theory to calculate the probability of pathways independent of contamination to produce burn wound infections. Results: Pathways independent of contamination may produce burn wound infections. The probability of certainty of this observation is 99.9998%. Conclusion: Pathways independent of contamination may produce burn wound infections.Scientific observations suggest pathways independent of contamination may produce infections [1,2]. For instance, unicellular organisms (prokaryota) including bacteria transformed from lifeless matter 3.5 billion years ago [3-5]. Murchison meteorite contain organic compounds consistent with the existence of vital substances necessary for life in universe [6] Organic compounds were produced by artificial methods [7].Microorganisms transform to other microorganisms [8]. Human cells transform to different cells [9,10]. A lifeless protein transforms to an infectious prion [11]. Tinea versicolor is caused by microorganisms that are part of normal flora [12,13]. Any one of the three host properties (genetics, medications, skin conditions) determines whether someone develops tinea versicolor [14,15] and this observation is consistent with a cell mediated model in which transformation can be ascribed. Also, studies indicate tinea versicolor is not contagious and it's not possible to infect a rabbit or human host with tinea versicolor by inoculating pityrosporum orbiculare or pityrosporum ovale without occlusion [14,15]. Together, these results describe a cell mediated model, in which transformation can be ascribed. H. pylori are part of normal gastric flora and result either from contamination or transformation. Epidemiological studies and clinical observations show no evidence of host to host contamination in h.pylori infections [16]. This observation is also consistent with a cell mediated model in which transformation can be ascribed.
Authors and Affiliations
Alen J Salerian
Keywords
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- EP ID EP591059
- DOI 10.26717/BJSTR.2018.08.001701
- Views 160
- Downloads 0
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